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Rhythm disruption promotes transformation by driving the loss of APC heterozygosity, which excessively activates Wnt signaling and MYC gene expression, consequently enhancing glycolytic metabolism, which is essential for sustaining tumor growth [202]
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Senomorphics: Modulating the Senescence-Associated Secretory Phenotype (SASP) Unlike senolytic therapies that aim to directly eliminate senescent cells, senomorphics work by modulating the senescence-associated secretory phenotype (SASP), which consists of pro-inflammatory cytokines, chemokines, growth factors, and extracellular matrix-degrading enzymes secreted by senescent cells
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