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Cyclization and PEGylation are established tools for prolonging peptide activity in the body
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A 2011 mechanism paper by Chang and colleagues in Journal of Applied Physiology demonstrated that BPC-157 promotes tendon fibroblast outgrowth, supports fibroblast survival under oxidative stress, and facilitates cell migration through the FAK-paxillin signaling pathway — a pathway central to cell adhesion and movement
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