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In rat HD models, neuropathological changes in the rat striatum were induced by 3-nitropropionic acid, and L-theanine exhibited neuroprotective effects, which mainly depended on not only inhibiting the production of harmful NO but also preventing the change of neurotransmitters in the striatum (51)

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When medical ozone contacts blood components, it creates controlled oxidative exposure that paradoxically reduces chronic oxidative stress over time by upregulating endogenous antioxidant systems: glutathione, superoxide dismutase, and catalase production increases in response