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The evidence is weak, because the required concentration of the thrombospondin-1 peptide exceeded the thrombospondin-1 concentration in plasma by 10 3 –10 4 (Lutz, 2013) and the phenomena had been studied on RBC that (a) were not actively leucocyte-depleted and (b) were oxidatively damaged by CuSO 4 and ascorbic acid rather than in vivo aged
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