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At the single-cell level, the lipid oxidation pathway in psoriatic keratinocytes is significantly upregulated compared to other cells such as fibroblasts, macrophages, dendritic cells, endothelial cells, and T cells, and it is highly associated with the Th22/Th17 pathway [206]

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A second mechanism is based on the competitive inhibition of the GSSG binding site by G-S-G and by the metal glutathione conjugate, as depicted in Scheme 11