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Maternal subcutaneous adipose tissues (ScAT) obtained from mothers with low circulating B12 showed upregulation of genes involved in the biosynthesis of cholesterol, such as 3-hydroxy−3-methylglutaryl-CoA reductase (HMGCR), sterol regulatory element binding protein 2 (SREBF2), low-density lipoprotein receptor (LDLR) and sterol regulatory element binding protein 1 (SREBF1) [40]