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Additionally, deoxyschisandrin suppressed H2O2-induced NF-kappaB activation by inhibiting IkappaBalpha degradation and NF-kappaB translocation to the nucleus, suggesting its potential as a protective agent against oxidative stress-induced intestinal cell apoptosis [481]
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We examined whether Nrf1 and Nrf2 are important in the transcriptional regulation of the rat GCLC using Nrf1 and Nrf2 knockout (F1 and F2) and wild-type (WT) fibroblasts and found that Nrf1 and Nrf2 are important for the rat GCLC promoter activity despite absence of ARE
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