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Decreased function of these enzymes, through genetic variability or presence of environmental chemicals, can lead to eventual interference with thyroid hormone, estrogen, and androgen levels [104, 105], as well as variable polyphenol effects [106], since the active forms of these compounds can be degraded via sulfonation

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Consistent with this, the NMDA receptor antagonist ketamine acutely activates mTORC1 signaling in mouse neurons, which coincides with an increased translation of synaptic proteins (Li et al., 2010)